EXAMINATION AND TREATMENT OF PERIPHERAL VESTIBULAR DISORDERS, UPDATED Final Examination Questions Select the best answer for each question and complete your test online at EliteLearning.com/Book 270. The semicircular canals of the vestibular system detect
277. Which of the following collective exam findings differentiates central vestibular pathology from peripheral pathology? a. Direction-changing nystagmus, suppressed with visual fixation. b. Direction-changing nystagmus, enhanced with visual fixation. c. Nystagmus that follows Alexander's law, enhanced with visual fixation. d. Central-gaze nystagmus, suppressed with visual fixation. 278. In the acute stage of vestibular dysfunction, spontaneous nystagmus that is found with gaze center, decreased with gaze toward the lesioned side and increased with gaze toward the unaffected side, is considered to be: a. Third-degree nystagmus. b. Indicative of a central lesion. c. Consistent with Alexander's law. d. Gaze-holding nystagmus. 279. A patient reports recent history of episodic vertigo while on vacation when coming up from the bottom of the pool or flying. What would be the most appropriate test to include in the clinical exam? a. Application of pressure to external ear canal. b. Hallpike-Dix test. c. Caloric test. d. DVA test. 280. Upon oculomotor testing of a patient with dizziness, you find corrective saccades on testing smooth pursuits and saccades. How would you manage this patient's findings? a. Prescribe gaze stabilization exercises. b. Refer the patient for further diagnostic workup. c. Prescribe habituation exercises. d. Perform an in-depth balance examination. 281. A corrective saccadic eye movement with a HIT to the right is interpreted as positive for: c. Left vestibular hypofunction. d. Right vestibular hypofunction. 282. A patient with a recent inner ear infection describes dizziness and blurred vision with walking and head turns. Which of the following tests is most appropriate to include in the clinical examination? a. Hallpike-Dix test. b. The MSQ. c. The DVA. d. The BBS. 283. A 3-line degradation on a DVA test is a clinical finding for which of the following conditions? a. BPPV. a. Left lateral canal BPPV. b. Right lateral canal BPPV.
what type of motion? a. Angular velocity. b. Linear velocity. c. Linear acceleration. d. Rotational acceleration.
271. In a normally functioning vestibular system, how will the neural firing rate change with head rotation to the right at 100 degrees per second? a. Increase on the left side by 100 degrees per second. b. Decrease on the left side by 50 degrees per second. c. Decrease on the right side by 50 degrees per second. d. Increase on the right side by 100 degrees per second. 272. The phenomenon of inhibitory cutoff occurs with head velocities greater than: a. 90 degrees per second. b. 180 degrees per second. c. 220 degrees per second. d. 550 degrees per second. 273. Which of the following are the gravity-sensitive structures of the vestibular system? a. Labyrinths. b. Otoliths. c. Semicircular canals. d. Ampullae. 274. With head rotation to the right, the MLF and ascending tract of Deiters mediates gaze stabilization through: a. Excitation of bilateral medial rectus muscles and inhibition of bilateral lateral rectus muscles. b. Excitation of right medial and left lateral rectus muscles and inhibition of right lateral and left medial rectus muscles. c. Excitation of medial and lateral rectus muscles on the right and inhibition of lateral and medial rectus muscles on the left. d. Excitation of left medial and right lateral rectus and inhibition of left lateral and right medial rectus muscles. 275. The primary symptom of benign paroxysmal positional vertigo is: a. Nonfatiguing nystagmus. b. Vertigo provoked with changes in head position. c. Vertigo induced by changes in pressure. d. Persistent vertigo at rest. 276. What is the mechanism that underlies spontaneous nystagmus in unilateral vestibular hypofunction? a. Reduced neural input to the ipsilateral ocular muscles, resulting in slow deviation eye movement toward the affected side. b. Reduced neural input to the contralateral ocular muscles, resulting in slow deviation eye movement toward the affected side. c. Increased neural input to the ipsilateral side, resulting in fast corrective movement toward the affected side. d. Increased neural input to the contralateral side, resulting in fast phase corrective eye movement toward the affected side.
b. Meniere's disease. c. Perilymph fistula. d. Vestibular hypofunction.
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